https://www.selleckchem.com/products/cc-99677.html
Mouse-adaptive mutations in the RV-A16 2C protein allowed for robust ****tion in cells expressing mSTING, suggesting a role for 2C in recruiting STING to RV-A ****tion organelles. Palmitoylation of STING was not required for RV-A16 ****tion, nor was the C-terminal tail of STING that mediates IRF3 signaling. Despite co-opting STING to promote its ****tion, interferon signaling in response to STING agonists remained intact in RV-A16 infected cells. These data demonstrate a surprising requirement for a key host mediator of inn
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