https://www.selleckchem.com/products/gsk343.html
G1 cells are overwhelmed by glycolytic intermediate accumulation due to failing mitochondria, while the functional electron transport chain and improved redox in G2 compensate for Warburg disruption. Both metabolomes reveal the production and abundance of epigenetic modifiers. G1 and G2 metabolomes differ and eventually determine the MDS clinical phenotype, as well as the potential for malignant transformation.Multi-omics signatures of patients with bladder cancer (BC) can guide the identification of known de-risked therapeutic compounds
Like
Comment
Share
