https://www.selleckchem.com/sc....reening/kinase-inhib
The acute phase response (APR) to CNS insults contributes to the overall magnitude and nature of the systemic inflammatory response. Aspects of this response are thought to drive secondary inflammatory pathology at the lesion site, and suppression of the APR can therefore afford some neuroprotection. In this study, we examined the APR in a mouse model of traumatic spinal cord injury (SCI), along with its relationship toneutrophil recruitment during the immediate aftermath of the insult. We specifically investigated the
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