https://www.selleckchem.com/products/nb-598.html
Based on the loss-of-function experiments, silencing expression significantly inhibited malignancy progression. In terms of the mechanism, acted on the ( ) by direct binding, which promoted BC cell growth. Furthermore, in the promoters of , the trimethylation of could be regulated by as the mediator. Relying on the / / pathway, the lncRNA was significantly associated with BC development and could, therefore, be a potential therapeutic target to block BC growth. Relying on the LL22NC03-N64E9.1/EZH2/KLF2 pathway, the lncRNA LL22NC03-N64E9.
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