https://alkpathway.com/looking....-at-probabilistic-ne
DG's mechanism of action involved upregulating FXR and SHP expression and downregulating hepatic de novo lipogenesis genes like SREBP1C, ACC1, and FASN. Additionally, DG fostered the expression of peroxisome proliferator-activated receptor alpha (PPAR), a key regulator of fatty acid breakdown. Besides this, DG blocked the expression of the CD36 molecule, contributing to fatty acid ingestion. Still, the inactivation of hepatic FXR lessened the regulatory impac
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