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Phosphatidylinositol 4-phosphate [PI(4)P] and phosphatidylinositol (34,5)-trisphosphate [PI(34,5)P3] levels are modulated by PIPkinA, which, consequently, influences the processes of phagocytosis and cell proliferation. Normal AprA binding is observed in cells lacking PIPkinA, suggesting that PIPkinA orchestrates chemorepulsion at a juncture subsequent to AprA receptor engagement and prior to AprA's repression of Ras activation. Bone marrow failure is a clinical h
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