https://www.selleckchem.com/products/apatinib.html
uced C3 and C5b-9 levels. These results suggest that oxidative stress-induced, complement activation-associated endothelial injury occurs in TA-TMA and that upregulation of Nrf2 protects endothelial cells from damage. Activation of the Nrf2 pathway may be a potential target for the treatment of complement activation-associated endothelial injury in TA-TMA.Limited information is available on the efficacy of post-transplantation cyclophosphamide (PTcy) or thymoglobulin for graft-versus-host disease (GVHD) prophylaxis in mismatched unrela
پسند
تبصرہ
بانٹیں
