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Along these lines, 11 obstructed the formation of F-action belts and the disintegration of bone in a concentration-dependent fashion. 11's mechanistic action decreased the expression of osteoclastogenesis-required genes by obstructing the translocation of NFATc1 from the cytoplasm to the nucleus. Furthermore, the action of 11 resulted in a therapeutic inhibition of human iPSC-sourced primary osteoclast differentiati
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