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A Western blot study demonstrated the expression of AP-1 in the nasal mucosa. Post-modeling, a significant elevation was observed in the AR symptom score, serum IgE and IL-4 levels, and the expression of TLR4, MyD88, and AP-1 proteins in the nasal mucosa of the model group, relative to the normal group. A substantial decline in serum IFN- content occurred simultaneously with the