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A consequence of ovarian cancer was a low level of PDLIM2 expression in tissues and cells. Attenuation of ovarian cancer cell proliferation, colony formation, migration, invasion, and epithelial-mesenchymal transition (EMT) was observed following PDLIM2 upregulation, along with inactivation of the TGF-/Smad signaling pathway. In PDLIM2-silenced ovarian cancer cells, the outcomes were precisely the reverse. Application of exogenous TGF- counteracted the suppression exerted by PDLIM2
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