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Prior research demonstrated a functional interplay between the FKBP52 immunophilin and tau, with a notable reduction in AD brain neurons correlating with tau accumulation. In AD neurons, FKBP52 was found to co-localize with autophagy-lysosomal markers and an early pathological variant of tau, thus suggesting its potential participation in autophagic tau clearance. We aimed to detect disparities in neuronal FKBP52 expression l
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