https://www.selleckchem.com/
Preservation of BD-donor hearts with CM resulted in a recovery of systolic graft function (dP/dtmax BD+CM 3148±178 vs BD2192±94mmHg/s at 110µl, p less then 0.05) and reduced apoptosis. LY294002 partially lowered graft protection afforded by CM in the BD-group. Our data suggest that PI3K/Akt-pathway is not the primary mechanism of action of CM in improving posttransplant cardiac contractility and preventing caspase-independent apoptosis. This article is protected by copyright. All rights reserved.PURPOSE To assess whether dietary intake of antioxidants, frui
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