https://www.selleckchem.com/pr....oducts/talabostat.ht
Transcriptome data further indicate that elevated expression levels of enzymes modulating VEGF-A availability were significantly connected to a decreased survival in bladder cancer patients. In comparison to previously postulated molecular players, we identified tumor cell derived VEGF-A and endothelial VWF as procoagulant mediators in bladder cancer. Therapeutic strategies that prevent the VEGF-A mediated release of VWF may reduce tumor-associated hypercoagulation and metastasis in bladder cancer patients. Implications We identified
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