https://nsc185058inhibitor.com..../compelling-children
Subsequently, our data revealed that S100A16 led to the destabilization of the LATS1 protein, facilitated by CUL4A-mediated ubiquitination and proteolytic degradation. This study highlighted the critical biological role of S100A16 in glioma progression, facilitated by its promotion of CUL4A-mediated LATS1 ubiquitination, thus hindering the Hippo signaling pathway. Glioma patients may see S100A16 as a novel biomarker and a prom
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