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All rights reserved. For permissions, please e-mail journals.permissions@oup.com.[This corrects the article DOI 10.1371/journal.pone.0221750.].Opioid addiction is recognized as a chronic relapsing brain disease resulting from repeated exposure to opioid drugs. Cellular and molecular mechanisms underlying the ability of organism to return back to the physiological norm after cessation of drug supply are not fully understood. The aim of this work was to extend our previous studies of morphine-induced alteration of rat forebrain cortex protei
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