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In this review, the mechanisms by which sodium-glucose co-transporter 2 inhibitors might protect the kidneys are examined, including their influence on tubuloglomerular feedback, albuminuria, endothelial function, erythropoiesis, uric acid levels, renal oxygen demand, and hypoxia. Moreover, we delve into the outcomes of current extensive clinical studies utilizing sodium-glucose co-transporter 2 inhibitors in individuals with c
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