https://nur77signal.com/index.....php/somatotopic-firm
MutPred2 established the candidate variant's pathogenicity via the loss of both phosphorylation and sulfation as actionable hypotheses. According to Project HOPE, the identified variant residue has a smaller size and a higher degree of hydrophobicity in comparison to the wild-type residue. I-TASSER and UCSF Chimera were employed for modeling and visualizing the predicted variant, respectively. A novel DLX3 variant implicated in
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