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burnetii remains viable but non-****tive within the host phagolysosome, as co-infection with C. burnetii expressing native EirA rescues the ****tive defect in the mutant strain. In addition, while the bacterial ultrastructure appears intact, there is an altered metabolic profile shift in the absence of EirA, suggesting that EirA may impact overall metabolism. Most strikingly, in the absence of EirA, Dot/Icm effector translocation was inhibited even when EirA deficient C. burnetii ****ted in WT supported Coxiella-containing vacu
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