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We propose that B cells contribute to late rejection as antigen-presenting cells for intragraft memory T cell expansion but not to alloantibody production and that a therapeutic strategy combining donor apoptotic cells, anti-CD40L, and rapamycin effectively inhibits proinflammatory B cells and promotes long-term islet allograft survival in such recipients. © 2020 The American Society of Transplantation and the American Society of Transplant Surgeons.OBJECTIVE To validate the performance of a first-trimester simple risk score based on t
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