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Similarly, an upsurge in SEMA3A and NRP1 expression was observed within HMVE cells, despite the absence of growth inhibition. The presence of VPA-induced SEMA3A in the OS cell culture environment diminished vascular tube formation in HMVE cells; further, escalating SEMA3A expression intensified the suppression of OS cell growth. The G1/S cell cycle arrest observed in OS cells was a direct result of the growth-inhibitory action exerted by SEMA3A. Via autocrine a
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