https://www.selleckchem.com/pr....oducts/Ilginatinib-h
felis colonization compared to infected TGF-β mice. In a T cell-deficient background using TGF-β xRag1KO mice, H. felis colonization was significantly lower when DC-derived TGF-β was absent, revealing a direct, innate function of DC in controlling H. felis infection independent of Treg induction. Our findings indicate that DC-derived TGF-β mediates Helicobacter-induced Treg response and attenuates the inflammatory Th1 response. We also demonstrated a previously unrecognized innate role of DC controlling Helicobacter co
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