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Local iron overload's impact on DSCs resulted in ferroptosis through a reduction in the concentrations of glutathione (GSH) and glutathione peroxidase 4. By supplementing with glutathione (GSH) and cysteine (used in the synthesis of GSH), iron-induced lipid reactive oxygen species (ROS) and cell death in differentiated stem cells (DSCs) were reduced. In models of spontaneous and LPS-induced abortion, ferroptosi
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