https://baf-a1inhibitor.com/cl....inicopathological-re
Due to the deletion of Acot12, a surplus of LPA is generated. This excess LPA signals through LPA receptors (LPARs) that are associated with G12/13 subunits, thereby hindering YAP phosphorylation and promoting YAP's nuclear localization and transcriptional action. The findings demonstrate Acot12's protective action against hepatocarcinogenesis, achieved by limi
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