https://www.selleckchem.com/products/myci361.html
indicated that hepatic ETAR knockdown may be a promising therapeutic strategy for metabolic diseases. This study defined an ameliorative role of hepatic ETAR knockdown in HFD-induced metabolism abnormalities by alleviating p66Shc-mediated mitochondrial fragmentation and consequent oxidative stress-related disorders and indicated that hepatic ETAR knockdown may be a promising therapeutic strategy for metabolic diseases. To study the risk factors of gestational diabetes mellitus (GDM) heterogeneity, and to evaluate the correlation between
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