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The most significant partial haplotype linked with the unsolved CVID was W*010101-DMA*010101-DMB*01030102-TAP1*010101 (P less then 0.001), where carriers had a late onset of the disease, only infection clinical phenotype, a non-familial form of CVID, post-germinal center defects and a non-progressive form of their disease. Exclusion of monogenic diseases allowed MR analyses to identify significant genetic variants associated with bacterial infections and improved discrepancies observed in MR analyses of previous GWAS studies with low ple
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