https://pafrsignaling.com/inde....x.php/ciceros-demarc
Additionally, the MEK/ERK signaling pathway was activated by GPR65 to orchestrate the proliferation of BCSCs. Finally, in vivo animal studies substantiated that a reduction in DSCR9 hindered BCSC proliferation, a phenomenon reversed by increasing GPR65 expression. By studying DSCR9's effects, we discovered that this molecule elevates GPR65 expression via its influence on miR-504-5p, ultimately worsening breast cancer. This breakthrough suggests new directions in
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