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In comparison to control groups, elevations in CK-MB and cTnI were observed. Ejection fraction and fractional shortening showed a decrease (001). A rise in the internal end-diastolic and end-systolic dimensions of the left ventricle was quantifiable. The model group demonstrated an increase in inflammatory markers, including IL-2, IL-6, TNF-, and hs-CRP. Inflammation of the myocardium, coupled with damage, was further confirmed by HE staining. QS's influence
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