https://www.selleckchem.com/pr....oducts/Acetylcholine
These results also open perspectives for understanding the epigenetic/genetic background of CML predisposition and for developing new therapeutic strategies.Therapy of malignant glioma depends on the induction of O6-methylguanine by the methylating agent temozolomide (TMZ). However, following TMZ exposure, most glioma cells evade apoptosis and become senescent and are thereby protected against further anticancer therapy. This protection is thought to be dependent on the senescent cell anti-apoptotic pathway (SCAP). Here w
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