https://www.selleckchem.com/products/jhu-083.html
Both history of early-life stress (ELS) and female **** are associated with increased risk for depression. The complexity of how ELS interacts with brain development and **** to impart risk for multifaceted neuropsychiatric disorders is also unlikely to be understood by examining changes in single genes. Here, we review an emerging literature on genome-wide transcriptional and epigenetic signatures of ELS and the potential moderating influence of ****. We discuss evidence both that there are latent **** differences revealed by ELS and that