https://jnj-26481585inhibitor.....com/bacteria-through
DG's mechanistic role included the upregulation of FXR and SHP expression and the downregulation of hepatic de novo lipogenesis genes, including SREBP1C, ACC1, and FASN. DG, consequently, stimulated the expression of peroxisome proliferator-activated receptor alpha (PPAR), a protein fundamentally involved in the process of fatty acid oxidation. Additionally, DG prevented the expression of the CD36 protein, essential for the process of fatty acid uptake.
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